You should go back to your doctor and discuss alternatives. Ask your doctor if the furosemide and losartan combination is suitable for you (taken separately with a few hours gap).

“Device-Guided Breathing to Lower Blood Pressure: Case Report and Clinical Overview” by William J Elliot reports positive results, but “Effect of device-guided breathing exercises on blood pressure in hypertensive patients with type 2 diabetes mellitus: a randomized controlled trial.” reports no significant difference.

The device is supposed to control your breathing to 10 breaths per minute, but according to the second study I mentioned, 40% of people did not achieve this.

I would certainly try controlling my breathing without a device before spending money on one.

The bigger issue, is whether controlled breathing will actually affect blood pressure. There are many causes of high blood pressure, and it would be irresponsible to try it without proper medical consultation.

Whether these results would apply to individuals with long-established hypertension or to people with “normal” serum urate values are questions that deserve answers. :

This article is exactly my response to this report, a while back.
I have wondered the same, especially as young people will not have the negative co- factors that older people generally tend to have [I won't list them].
The treatments for Hi BP are well known for only partial effectiveness (Ive seen 30% full success ‘back to normal’ mentioned) against this remarkably high result.
Whether this effect can be sustained longterm is another matter.
BP. meds. are notorious, also ,for losing their effectivness over time, and then being rested/restarted later- or switched to another type to maintain some relief from the BP going high or their side effects.
My hopes are quite high on this work being followed up- but I still resist adding more meds to my armoury- Yet!

Given these observations, the long-appreciated association
between hyperuricemia and hypertension begins to take on
an appearance of cause-and-effect. The question logically
follows: might lowering serum urate provide a therapy for
essential hypertension? Johnson’s group performed a small,
open-label proof of principle study to test that hypothesis.
They enrolled five adolescents with documented essential
hypertension, all with serum urates of 6.0 mg/dL or above,
in a 10-week trial. For the first 4 weeks, all of the patients
received allopurinol, 200 mg BID. The remaining 6 weeks
were a washout period. Blood pressure was measured before
the administration of allopurinol, at the end of the four-week
treatment course, and at the end of the washout period. After
4 weeks of allopurinol therapy, all five patients demonstrated
lowered blood pressure, with four of the five in the normal
range. After the washout period, all five had significant
increases of their blood pressures back toward their initial
starting values.21 A larger blinded study is ongoing. These
data suggest that urate can positively regulate human blood
pressure, and that interventions to lower urate may be of
value. Whether these results would apply to individuals with
long-established hypertension or to people with “normal”
serum urate values are questions that deserve answers.

http://74.125.77.132/search?q=cache:gv73IAeydXwJ:ftp.rta.nato.int/public//PubFullText/RTO/TR/RTO-TR-014///TR-014-S4-04.pdf+uric+acid+reduction+with+lasix+losarten&cd=7&hl=en&ct=clnk

Quote page2 : While up to 120 mg of losartan has little measurable effect
in salt-replete individuals, the systemic pressor response to angiotensin II is blocked,
from a threshold effect at 10-20 mg to a maximal effect at 80-120 mg.

In normals who were salt-depleted by
a low sodium diet plus furosemide, losartan induced
a dose-related fall in systolic and diastolic blood pressure.

malik says: uric acid control

What does that mean?

Are you seeking uric acid control, offering it, or advising it?

Do you know something about uric acid and blood pressure medication that you can share with the rest of us?

If this is such a useful protocol- and I hope it works for me, it’s possible half doses taken twice daily, with the requisite gap between them, could be doubly helpful too!
Both tablets are ’splittable’ , btw.

Well, I ‘ve got the go ahead to try this combo.

Out of interest, did this need persuasion, or was you doc already aware of the benefits of this combo?

I have read that furosemide alone is only 25% as likely to exacerbate gout than thiazide diuretics (most commonly hydrochlorothiazide.) That prompted me to switch several years ago. The major difference between the thiazides and the loop (Lasix) is the location of action in the kidney. The thiazides dump copious sodium and the water that follows it, the loop diuretic dumps WATER. (If you taste your urine the difference is astounding.)
It seems the loss of sodium cannot be accomplished without the retention of urate.

A sidebar here is that on thiazides I ALWAYS ran severely low sodium levels, to the point where doctors were concerned I was still standing…my sodium remains perfectly normal on Lasix (furosemide…the loop diuretic.)

So the glib condemnation of diuretics in learned tomes on gout is only half the story.

When I read that losartan was uricosuric, I added it hoping to at least keep my uric acid stable. What a joy to find that these kindly considerate Japanese researchers had found a way to do even better…to LOWER my uric acid.

Someday when I am brave, I will try a regimen of only the Lasix followed later by Cozaar…and stop my allopurinol for a couple months. I’d have done it already but for the presence of a single tophus on my little finger. But I so dread an acute attack, pussy that I am!.

The lasix-cozaar combo gives me very good BP control…<130/<75

I, and most of my readers, have little or no medical knowledge.

I haven’t a clue how this relates to the losartan-wait-furosemide treatment.